1. Types of ion channels found on nerve cells: 

• Voltage-gated channels 
• Ligand-gated channels

2. 3 differences between Voltage-gated channels and Ligand-gated channels:

Voltage-gated channels	Ligand-gated channels
Changes in membrane potential of the cell.	Binding of ligand to ion channel.
Transmits signal from the cell body to the nerve terminal.	Can be regulated by multiple mechanisms including phosphorylation and endocytosis.
Works with Sodium, Potassium and Calcium channels.	Very rapid action between binding of an agonist to the ligand-gated channel and a cellular response.

3. Comparison of ionotropic and metabotropic receptors: 

Ionotropic	Metabotropic
Ligand-gated ion channel receptors	7-Transmembrane G-protein coupled
Post synaptic potential	Production of second messengers that modulate ion channels
Works on multiple ion channels	Effects last longer as compared to ionotropic receptor activation.
Opening of ion channels	Metabolic changes
	G-protein dependant receptors

4. Classify CNS receptors into inotropic and metabotropic:

Ionotropic: 

• GABA_A ( g-amino butyric acid)
• Nicotinic (Acetylcholine)
• EAA (Glutamate)
• 5-HT₃ (Serotonin)

Metabotropic:

• Adenylyl cyclase system
• Phospholipase C system 

5. Difference between EPSP and IPSP: 

EPSP, excitatory post synaptic potentials-An electrical change such as depolarisation in the membrane of a postsynaptic neuron caused by the binding of an excitatory neurotransmitter from a presynaptic cell to a postsynaptic receptor thus making it more likely for a postsynaptic neuron to generate an action potential.

This is seen by, the Serotonin receptor causing depolarization in the sodium ion channel. 

IPSP, inhibitory postsynaptic potentials (IPSPs): a hyperpolarizing current that causes the membrane potential to become more negative.

This is seen with GABA receptors being hyperpolarized when opening of chloride channels.

6. Role of calcium in the development of synaptic potential: 

The release of a synaptic potential is dependant of calcium